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Spinal Cord Injuries, pp. 483-499 $100.00
Authors:  Kasim Abul-Kasim, Anita Strömbeck and Pia C Sundgren
Spinal cord injury can be classified as traumatic and non-traumatic. The traumatic spinal
cord injuries (SCI) are caused by motor vehicle accidents (56 %), falls (14 %), firearm and
violence-related (16.6 %) and sports injuries (7 %) [1]. Injuries after falls and minor trauma
are more commonly seen in elderly patients as they more often have spondylosis and
osteoporosis. Violence is more common in urban populations while sports injuries are
common in young individuals. About 68 % of children involved in spinal cord injuries
caused by motor vehicle accidents were not wearing a seatbelt. Almost 80% of patients with
spinal cord injury had multiple injuries [2]. Associated injuries include other bone fractures
(29.3 %) and brain injury (11.5 %) [3]. Other causes of spinal cord injuries are non-traumatic
and include the following: vascular disorders, degenerative disorders, spinal tumors,
infectious and inflammatory conditions of the vertebral column with secondary SCI as well
as iatrogenic injuries after spinal injections and epidural catheter.
Three possible mechanisms are believed to be involved in the development of spinal cord
injuries [4]: (a) damage from direct trauma, (b) compression or transaction of neural elements
by bone fragments, intraspinal hematoma, foreign bodies, or protruded disk, or (c) ischemia
from damage of the spinal arteries or from venous congestion. As small arteries are disrupted
by trauma, spinal cord swelling occurs within minutes after the trauma with resultant venous
congestion and secondary ischemia. Cell death occurs days to weeks after the injury with
involvement of the oligodendrocytes not only at the site of injury but also at several levels
away from the injury site [5]. Following the primary spinal cord injury, a cascade of
secondary injuries usually are initiated [6] resulting in: (a) vascular changes—ischemia,
hemorrhage, and thrombosis [7], (b) Disturbance of electrolyte balance with accumulation of intracellular sodium resulting in edema [8], (c) accumulation of neurotransmitters and toxins
edema [8], (d) inflammation [9], and (e) apoptosis. 

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Spinal Cord Injuries, pp. 483-499