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Circadian Dysrhythmia-Associated Type 2 Diabetes and Chrononutrition (pp. 39-42) $0.00
Authors:  Pradeep Visen, Aastha Visen, Srishti Visen, and Asha Sharma
In mammals, the circadian timing system drives rhythms of physiology and behavior, including the daily rhythms of feeding and activity. Circadian disruption (e.g., as a result of shift work or jet lag) can disturb this continuity and increase the risk of cardio-metabolic disease. Recent researches are providing link and evidences that circadian rhythm dysfunction might play an important role in the etiology, course or prognosis of type 2 diabetes. An interest has thus emerged in the development of novel clock-targeting pharmaceuticals or nutraceuticals for the treatment of metabolic dysfunction. Recently, the pineal hormone melatonin has received some attention as a potential chronotherapeutic drug for metabolic disease. Melatonin is well known for its sleep-promoting effects and putative activity, stimulating coordination of biochemical chain through targeting the internal timing system. Melatonin affects the insulin secretory activity of the pancreatic beta cell, hepatic glucose metabolism and insulin sensitivity. Reduced mela-tonin levels, and mutations of melatonin receptors are associated with an increased risk of developing type 2 diabetes.
In this presentation, we briefly review our understanding of molecular clock control of glucose homeostasis, influence of circadian disruption on glucose metabolism and other macronutrients, explore the contribution of melatonin signalling to the etiology of type 2 diabetes and its role in disease management. 

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Circadian Dysrhythmia-Associated Type 2 Diabetes and Chrononutrition (pp. 39-42)