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01.Focus on Leukemia Research (Horizons in Cancer Research, Volume 5)
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03.Advances in Neonatal Hematology
04.Blood Infections: Scientific, Clinical and Public Health Aspects
05.New Developments in Sickle Cell Disease Research
06.Thrombocytopenia: Causes, Diagnosis and Treatment
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08.Deep Vein Thrombosis: Symptoms, Diagnosis and Treatments
09.Focus on Sickle Cell Research
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New Developments in Sickle Cell Disease Research
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Book Description:
Sickle cell anemia is an inherited blood disorder, characterized primarily by chronic anemia and periodic episodes of pain and occurring in approximately 1 in every 400 African-American infants born in the United States each year. Individuals of Mediterranean, Arabian, Caribbean, South and Central American, and East Indian ancestry can also be affected. The underlying problem involves hemoglobin, a component of the red cells in the blood. The hemoglobin molecules in each red blood cell carry oxygen from the lungs to the body organs and tissues and bring back carbon dioxide to the lungs. In sickle cell anemia, the hemoglobin is defective. After the hemoglobin molecules give up their oxygen, some of them may cluster together and form long, rod-like structures. These structures cause the red blood cells to become stiff and to assume a sickle shape. Unlike normal red cells, which are usually smooth and donut-shaped, the sickled red cells cannot squeeze through small blood vessels. Instead, they stack up and cause blockages that deprive the organs and tissue of oxygen-carrying blood. This process produces the periodic episodes of pain and ultimately can damage the tissues and vital organs and lead to other serious medical problems. Unlike normal red blood cells, which last about 120 days in the bloodstream, sickled red cells die after only about 10 to 20 days. Because they cannot be replaced fast enough, the blood is chronically short of red blood cells, a condition called anemia. Sickle cell anemia is caused by an error in the gene that tells the body how to make hemoglobin. The defective gene tells the body to make the abnormal hemoglobin that results in deformed red blood cells. This book gathers the latest research in this important field.

Table of Contents:
Preface;

1. Malaria Resistance or Susceptibility in Red Cells Disorders, pp. 1-61;
(Farba Balle Khodia Faye, Institut de Recherche pour le Developpement, Senegal);

2. Resonant Raman Studies on Functional Erythrocytes, pp. 63-119;
(Bayden R. Wood and Don McNaughton, Monash University, Australia);

3. An Additional Copy of Chromosome 21 Drives Your Genes Wild, pp. 121-143;
(Aliza Amiel et al., Meir General Hospital, Israel);

4. Psychobiological Reactivity in Children with Sickle Cell Disease: A Novel Variable for Prediction of Outcomes and Improving Interventions, pp. 145-168;
(Marsha J. Treadwell, Children's Hospital and Research Center at Oakland, USA);

5. Acute Chest Syndrome in Sickle Cell Disease: Pathophysiologic Approach to Management, pp. 169-192;
(Steven H. Yale, and Joseph J. Mazza, Marshfield Clinic, USA);

6. A Decade of Advances in Sickle Cell Disease Management, pp. 193-210;
(Allan Platt, Melanie Jacob and James R. Eckman et al., Emory University School of Medicine, USA);

7. The Heart in Sickle Cell Disease, pp. 211-226;
(Athan Aessopas);

8. Oral Health Status and Anthropometric Findings in Sickle Cell Disease, pp. 227-240;
(F.A. Oredugba and K.O. Savage, University of Lagos, Nigeria);

Index

   Pub. Date: 2006
   ISBN: 978-1-59454-792-8
   Status: AV
  
Status Code Description
AN Announcing
FM Formatting
PP Page Proofs
FP Final Production
EP Editorial Production
PR At Prepress
AP At Press
AV Available
  
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New Developments in Sickle Cell Disease Research